Case of the Week #610

Frantisek Grochal (1); Vladimir Lemaire (2)
(1) Femicare, Center of prenatal ultrasonographic diagnostics, Martin, Slovakia; (2) UT Southwestern Medical Center, Plano, Texas, United States of America.

Posting Dates: Jul 31, 2024 - Aug 14, 2024

A 32-year-old primigravida presented to our office at 19w 2d for a second-trimester ultrasound examination. The following anomalies were seen:

Image 1
Image 2
Image 3
Video 1 © 2024 Frantisek Grochal
Video 2 © 2024 Frantisek Grochal
Image 4
Video 3 © 2024 Frantisek Grochal
Video 4 © 2024 Frantisek Grochal

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Answer

We present a case of Tetralogy of Fallot with Absent Pulmonary Valve Syndrome.

Our examination revealed the following findings:

  • left axis deviation of the heart;
  • ventricular septal defect with overriding aorta;
  • large main pulmonary artery with turbulent high velocity flow and marked dilatation of the right and left pulmonary arteries;
  • absent ductus arteriosus.

The patient was sent to the National Institute for Cardiovascular Diseases (Bratislava, Slovak Republic) where the diagnosis was confirmed. The parents opted for termination of the pregnancy.

Four-chamber-view of the heart with left axis deviation (RV: right ventricle, LV: left ventricle, LA: left atrium, RA: right atrium).
Image 1 Four-chamber-view of the heart with left axis deviation (RV: right ventricle, LV: left ventricle, LA: left atrium, RA: right atrium).
Ventricular septal defect with overriding aorta (LV: left ventricle, RV: right ventricle, Ao: aorta, VSD: ventricular septal defect).
Image 2 Ventricular septal defect with overriding aorta (LV: left ventricle, RV: right ventricle, Ao: aorta, VSD: ventricular septal defect).
Video 1 Left axis deviation and ventricular septal defect with overriding aorta. © 2024 Frantisek Grochal
Video 2 Dilated main pulmonary artery and its branches with turbulent flow. © 2023 Frantisek Grochal
Video 3 4D - Dilated main pulmonary artery and its branches. © 2023 Frantisek Grochal
Video 4 4D - Dilated main pulmonary artery and its branches, absent ductus arteriosus. © 2023 Frantisek Grochal

Discussion

Absent pulmonary valve syndrome (APVS), also referred to as tetralogy of Fallot (TOF) with absent pulmonary valve, is a rare cardiac malformation. It accounts for 3 to 6% of all patients with TOF and for 0.2 to 0.4% of live-born infants with congenital heart disease. APVS is characterized by absent, rudimentary, or dysplastic pulmonary valve leaflets associated with an outlet ventricular septal defect (VSD) and an overriding aorta. In most cases, APVS is associated with an absent ductus arteriosus. The main, right and left pulmonary arteries become severely dilated, while the main pulmonary valve annulus shows signs of stenosis with severe insufficiency.  The aneurysmal dilation of the pulmonary artery is a pathognomonic sign of APVS.

The four-chamber view may appear normal but may already show a dilated right ventricle, especially in late gestation due to volume overload from the insufficient pulmonary valve. The left ventricular outflow tract view demonstrates the malaligned VSD with the overriding aorta. The aortic root is not dilated in APVS, conversely to the classic TOF. The three-vessel-trachea view demonstrates massively dilated main, right and left pulmonary arteries. The ductus arteriosus is not found, as in most cases of APVS.

High velocities across the pulmonary valve annulus with to-and-fro flow are a sign of stenosis and insufficiency, and are characteristic findings on color and pulsed wave Doppler. On spectral Doppler evaluation, velocities around 250 to 300 cm/s are generally obtained across the main pulmonary artery valve in late gestation. Color Doppler may also show tricuspid valve regurgitation.

Associated cardiac findings include right aortic arch and absent ductus arteriosus. Extracardiac findings include a high association with chromosomal anomalies, primarily DiGeorge Syndrome. The APVS variant with a patent ductus arteriosus is rarely associated with chromosomal or extracardiac abnormalities. Bronchomalacia, due to bronchial compression by the dilated pulmonary trunk, is a common and serious association with APVS. However, this condition cannot be diagnosed prenatally.

APVS is associated with a poor prognosis and survival rates between 15 to 30% have been reported in some series. Findings that suggest a poor prognosis include cardiomegaly and marked pulmonary dilation.

Reference

Abuhamad, A, et al. "Absent Pulmonary Valve Syndrome". A Practical Guide to Fetal Echocardiography: Normal and Abnormal Hearts (4th edition). Singapore: Wolters Kluwer, 2022. pgs 439-447.

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